CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores mitochondrial homeostasis in dominant optic atrophy patient-derived iPSCs: Molecular Therapy - Nucleic Acids
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Descrição
Melatonin Repairs Osteoporotic Bone Defects in Iron-Overloaded
CRISPR/Cas9-mediated A4GALT suppression rescues Fabry disease
Understanding the molecular basis and pathogenesis of hereditary
Functional genomics and the future of iPSCs in disease modeling
Characterisation of a novel OPA1 splice variant resulting in
IJMS, Free Full-Text
Functional genomics and the future of iPSCs in disease modeling
Frontiers CRISPR/Cas9: implication for modeling and therapy of
Enhanced genome editing in human iPSCs with CRISPR-CAS9 by co
Melatonin Repairs Osteoporotic Bone Defects in Iron-Overloaded
CRISPR/Cas9-mediated A4GALT suppression rescues Fabry disease
JCM, Free Full-Text
Frontiers Establishing induced pluripotent stem cell lines from
Drosophila model to clarify the pathological significance of OPA1
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