RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
Por um escritor misterioso
Descrição
Surviving death: emerging concepts of RIPK3 and MLKL ubiquitination in the regulation of necroptosis - Karlowitz - 2023 - The FEBS Journal - Wiley Online Library
Epigenetic Silencing of RIPK3 in Hepatocytes Prevents MLKL-mediated Necroptosis From Contributing to Liver Pathologies - Gastroenterology
RIPK3-and MLKL-dependent necroptosis mediated by various stimuli.
Canonical versus noncanonical necrosome activation by three distinct
Schematic representation of necroptosis within ischemic milieu. After
RIP3 is an upregulator of aerobic metabolism and the enhanced respiration by necrosomal RIP3 feeds back on necrosome to promote necroptosis
Examination of RVA-induced necroptosis and apoptosis by confocal
Nuclear RIPK3 and MLKL contribute to cytosolic necrosome formation and necroptosis
Roles of RIPK3 in necroptosis, cell signaling, and disease
Opposite Effects of Apoptotic and Necroptotic Cellular Pathways on Rotavirus Replication
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